Coronary Circulation in Nonsmokers and Smokers
There is a strong relationship between cigarette smoking and the risk of cardiovascular events. Smoking influences negatively coagulation - fibrinolysis cascade at any level, although it acts primarily on those pathways that are most important for an effective clot formation: endothelium, platelets, and fibronogen. Moreover, coronary circulation is the target organ of the damage due to smoking exposure. Endothelial dysfunction is a main consequence of smoke compounds with significant changes in initiating physiologic coagulation process; platelet adhesiveness and aggregation increases as a result of smoking; and, finally, fibrinogen/fibrin framework strengthens clot thickness.Therefore, the whole coagulation cascade activates the thrombi fo
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Coronary Circulation in Nonsmokers and Smokers
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